The group was focused in the T2DM resolution mechanism after bariatric surgeries. This surgical procedures are implied in obesity and comorbidities resolution. We developed several researchs in animals experimental models. Thus, we designed different surgeries similar to human clinic and other new experimental procedures. Afterward, we redirected our studies to human clinic. We are trying to probe the similities between animal findings and the human consequences on these several comorbidities. We have probed several cellular consequences on pancreas after surgeries. Even, the consequences on enterohormones released during and after surgeries.
Hepatic gland has a main importance in the T2DM resolution and obesity. We are implied in the hepatic influences in these clinical processes.
-Experimental animal models of bariatric surgery
-Mechanisms of resolution of T2DM after surgical techniques in humans.
-Enterohormones and their impact on the homeostatic mechanisms of glycemic control.
-Hepatic damage linked to obesity. Involvement in the resolution of T2DM. Nonalcoholic fatty liver damage.
-Bariatric sugeries in human clinic. Clinical treatment of obesity.
-Experimental surgeries related to bariatric surgeries. Animal models, similar to those employed in human clinic and experimental models.
-Immunofluorescen techniques, molecular biology techniques (WB, multiplex).